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The antigen recognized by M37 is unknown, but it is present in bah-1 mutants and therefore cannot be BAH-1 [16].
This allows one to identify equivalent cells in heterozygotes and mutants and therefore allows target gene expression to be compared in these cells in the presence or absence of the SSTF.
We unambiguously demonstrated that cell death is indeed non cell-autonomous using targeted ablation in the presence of the R26YFP reporter allele, thus allowing us to show that a large number of apoptotic cells are not recombined in Foxg1Cre and Wnt1 Cre ablated mutants, and therefore cannot undergo cell death because of DTA expression.
The T3SS is not functional in hrpS mutants and therefore no Pst-encoded virulence effectors would be delivered into the plant cell [ 35, 36].
In theory, such a complex can form also with some p53 mutants, and therefore ER-alpha may potentially influence the transcriptional landscape of cells expressing mutant p53.
This difference most likely reflects the fact that cdc13-1 mrc1Δ exo1Δ mutants have more severe telomere capping defect than cdc13-1 exo1Δ mutands and therefore activate a stronger checkpoint signal.
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Four mAbs (HX44, HGP105, HGW7, HGY38) reacted with wt and CD4bs YU2 mutants but failed to bind the CD4i mutant, and therefore were assigned to the CD4i cluster.
It has been previously shown that expression of a bni1 construct lacking this BBD region is unable to rescue the synthetic lethality of a bni1Δ cla4Δ mutant, and therefore the transformed mutants exhibit a terminal phenotype similar to that of ste20Δ cla4Δ mutants [47].
Here, a larger leaf size probably reflects an earlier block to GSH synthesis in the mutant, and therefore an earlier reduction in bps1 signal synthesis.
But this will benefit all individuals in species i, not just the mutant, and therefore has no differential selective benefit to the individual that bears the mutation [ 77].
This mutation does not reverse the loss-of-function seen in the Y206F mutant and therefore provides evidence that Y206 is acting as a hydrogen bond donor.
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