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Exact(28)

Starch granules in sug-1 and sug-h mutants also displayed irregular shapes with round edges, whereas those in the wild type displayed polygonal shapes with sharp edges (Fig. 1p-r).

Mutants also displayed severe growth reduction, and newly formed leaves were crumpled and serrated.

yata mutants also displayed various eye defects, including irregular arrangement of ommatidia, black discoloration, and lens defects (Figure 3D 3I).

In addition, and very conveniently for humans, these mutants also displayed a reduced virulence represented by lower adhesion to epithelial cells [18], [19], [20].

Mutation of SSN8 resulted in increased accumulation of melanin and capsule, two well-known virulence factors of C. neoformans; however, the ssn8 mutants also displayed altered phenotypes in other cellular and physiological processes.

The homozygous Sp4 mutants also displayed significantly reduced PPI relative to heterozygous Sp4 mice which in turn have less PPI than wildtype siblings, consistent with a gene-dosage effect.

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Similar(32)

Loss-of-function mutant of these mutants also display an ABA-deficient phenotype (Kang et al., 2010; Kuromori et al., 2010, 2011).

dnrx and dnlg1 mutants also display ultrastructural defects ranging from abnormal active zones, misformed pre- and post-synaptic areas with underdeveloped subsynaptic reticulum.

S. cerevisiae ras2 mutants also display reduced invasive growth [54].

Interestingly, C. elegans rab-3 null mutants also display resistance to aldicarb with only mild locomotor defects [54].

These three mutants also display the strongest leakage phenotypes (Figure 4), confirming the importance of the dityrosine layer in establishing the permeability barrier of the spore wall.

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