Sentence examples for mutant would cause from inspiring English sources

The absence of Thr338 in p.T338M mutant would cause a decrease in the stability of this region.

If so, this could suppress the increased ER oxidation levels caused by ERO1* observed in Figure 4. 2) A prediction from the model is that GAL1 induced over-expression of ERO1* in the Kar2 C63A mutant would cause an accumulation of unfolded secretory proteins in the ER and/or translocation defects compared to the wild type Kar2 background.

As a result, this mutant would hardly cause negative consequence to MxA compared with the reported D478 mutant.

Acquisition of a p53 defect in this context would cause mutant cells to 'perceive' mitogenic signals as short despite ErbB2-dependent signal prolongation – leading to apoptotic resistance, dedifferentiation and clonal outgrowth.

Moreover, because ARF5/MP stimulates PIN1 expression and thus auxin efflux [ 46, 47], the undertranslation of ARF5 in the eif3h mutant (Fig. 6) would cause underexpression of PIN1 PIN1-GFP, as well as a tendency for PIN1 PIN1-GFPoots, as wellindeed observed (Fig. 4C, D; Fig. 3as.

Upstream regulators of B gene expression as UFO, LEAFY or AP1 are unlikely to be affected in the C. vulgaris bud bloomer mutants, because dysfunctions in these genes would cause severe flower malformations: UFO mutants in Arabidopsis display filamentous structures instead of flowers [ 35].

Of course, if global warming alarmists really had predicted that it would cause more frequent and severe cold outbreaks (via Mutant Polar Vortexes, Mutant Teenage Ninja Turtles, Yeti, or whatever), we should see such predictions all throughout the latest United Nations Intergovernmental Panel on Climate Change (IPCC) report.

However upon deletion of BLR1/BLR2 or ENV, both light induction and adaptation are assumed to be lost, which would cause decreased/altered transcript levels in the mutants.

However, analysis of the PLP cofactor binding and enzyme kinetics of these two mutant mimics does provide some clue as to why these changes would cause alterations of the hLCB2a subunit that would lead to the HSAN1 phenotype.

To test whether knockdown of gcm2 would cause similar hair cell defects as seen in merovingian mutants, we injected fish with a gcm2 MO.

That would cause a sensation.