Sentence examples for mutant which we from inspiring English sources

This mutant, which we denoted as long vegetative phase 1-1D (lov1-1D), flowithd with 26.6 leaves under long days, whereas wild-type plants flowered with 15.5 leaves (Figure 1A).

The photosensitive phenotype of our gun4-II mutant resembles that of the earlier identified Chlamydomonas gun4 mutant which we will refer from here onward, as gun4-I.

1) The akin 10 mutant: We understand the necessity of a comprehensive characterization of the akin10 mutant, which we used for our study, and we apologize for not including that in our manuscript.

For comparison, we studied another DGAT1 mutant, which we designated A7, which is a homozygous SALK line (Salk 039456) with a T-DNA insertion in the last exon of the same DGAT1 gene (At2g19450).

We have tried to improve the discussion of this mutant, which, we realize, is complex, as it entails the interaction with PP2A, the possibility that the deletion removes an important regulatory intramolecular interaction, etc.

Conversely, the R33Q mutant, which we previously found to be better phosphorylated than wild-type Parkin [ 26] due to presumed disruption of the Ubl domain autoinhibition, showed only a moderate change in phosphorylation by PINK1 upon addition of ubiquitinPhospho-Ser65 (Fig EV 4).

We found that in zebrafish erbb2 mutants, which we show completely lack cardiac trabeculae, cardiac function is significantly compromised, with mutant hearts exhibiting decreased fractional shortening and an immature conduction pattern.

The only significant difference phenotypically between the two mutants was the higher penetrance of abnormal teeth on the mandible in Wise mutants, which we attribute to the fact that the Lrp4 mutation is hypomorphic, whereas Wise is believed to be a null.

To determine whether Rad8 functionally overlaps with other helicases, we compared the phenotypes of single and double mutants with a panel of 23 nonessential helicase mutants, which we categorized into five phenotypic groups.

A strong signal was detected in siblings at 3 dpf that appeared to be markedly reduced in tmod4 trg mutants, which we believe is due to nonsense-mediated decay (Fig. 2E).

This is similar to the M404V/T UBA mutants which we have previously reported to be associated with poor expression levels (as UBA only sequences), lower solubility, and aggregation effects consistent with destabilisation of the UBA domain [24].