On the one hand, experiments utilizing fluorescence resonance energy transfer (FRET) measurements with fluorophore-tagged subunits and cysteine knockout mutants of GluN1 were interpreted in terms of a dimer of homodimers [5], [6].
No trends were interpreted.
Lastly, we also note that the observation of Geibel at al. that the blue light response changes its sign exclusively in the triple mutant could be interpreted as an indication that this mutant has altered the directionality of proton currents and that this would be compatible with our suggestion of back flow under conditions of positive potential.
Synthetic phenotypes produced by simultaneous depletion of two genes and not observed in either single mutant often are interpreted as an indication of functional connections between genes.
Since growth is a prerequisite for the membrane damage caused by carolacton treatment [ 5], the results of the Live/Dead staining for the fabT mutant should be interpreted with caution.
This means that positional information cannot be defined by any one factor in isolation, and nor can mutant results be interpreted reliably in the absence of data on other factors.
The loss in activity of EGS Lys132-mutants can be interpreted in terms of the proposed mechanistic model, in conjunction with results from structural analyses.
The lack of lifespan reduction in the mitochondrial SOD mutants has been interpreted as a major challenge to the mFRTA [12], [24], [28], [32], [33].
Since the viruses used in our study only differ in the gp120 V3 loop region, the enhanced binding to the various CXCR4-g1 mutants can be interpreted as an effect of the mutated coreceptors and the V3 loop.
However, the phenotypes observed in Δ UGH and Δ UGH/Δ URE (urease) deletion mutants have been interpreted according to the incorrect assignment of this DAL3 homolog to EC 3.5.3.19.
