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The number of seeds per ear (approximately 60 seeds) and ears per plant of the fpgs03g mutant were also similar to wild type.

The genes induced or repressed by MYCNΔMBII-ERTM mutant were also generally delayed in response compared to wt.

In addition to the two RNAi dystrophin mutants mentioned above, enhancers of the DysE6 mutant were also screened for.

Furthermore, in depth characterization of the quinolone resistance mechanisms as well as the whole process of becoming a high-level resistant mutant were also a matter of concern.

Likewise, the wild-type and the hcpA mutant were also used to study the induction of proinflammatory cytokines in different media.

As a control and a means of determining maximal possible inhibition of demethylase activity, cells expressing the JMJD2A H188A catalytically deficient mutant were also quantified in each experiment.

The locations of the fast inactivation particle (h gate) and the A1273V mutant are also shown.

Strikingly, the acra-acga- mutant was also not defective in fruiting body morphology or sporulation (Fig. 4A,B).

Interestingly, the overall catalytic efficiency (kcat/Km) of mutant was also enhanced (~ 20 folds).

Since acetate kinase is the enzyme that converts acetyl phosphate to acetate and ATP, an ackA mutant is also expected to be defective in anaerobic growth on xylose.

Activation of GA biosynthesis in the aba2-2 mutant was also observed during seed development.