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Upon ectopic expression of CsTFL1b in Arabidopsis, the flowering time of transgenic plants was significantly delayed in both wild type and tfl1-11 mutant background but the terminal flower phenotype of tfl1-11 mutant was partially rescued.
The T315I mutant was partially inhibited by dasatinib or GNF-5 in this assay.
As shown in Fig. 5A, the K189A rNSs mutant was completely inactive whereas the D159A mutant was partially active.
As above, the reduced biomass generated by the lrgAB mutant was partially complemented (8.69±0.813 µm3/µm2) by the presence of the lrgAB expression from a plasmid.
As shown in Figure 6A, alleviation of silencing in the mcl1 mutant was partially suppressed by multi-copy plasmids of sir2+ gene and, to a lesser extent, of the clr3+ gene.
Seven null mutants remained sensitive to GEL, whereas the growth of the ΔnemA mutant was partially recovered at 30°.
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Furthermore, alterations in cell wall composition in the crc knu mutant were partially rescued when YUC4 was misexpressed.
Jung, I. L., Oh, T. J. & Kim, I. G. Abnormal growth of polyamine-deficient Escherichia coli mutant is partially caused by oxidative stress-induced damage.
Meiotic defects of the nbs-1 mutant are partially suppressed by abrogation of the nonhomologous end-joining (NHEJ) pathway, indicating a role for NBS-1 in antagonizing NHEJ during meiosis.
These results suggest that the late flowering of the z3 mutant is partially due to the retardation of growth resulting from unbalanced distribution of metal ions.
The Rv3802 mutants of S175ΔE and S86ΔE were essentially inactive, while the S87ΔE mutant is partially active in both assays (Table 1).
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