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The lba1 mutant was originally isolated as a mutant exhibiting reduced sugar-induced expression of Atβ-Amy [52].
This mutant was originally engineered with the aim to mimic the catalytic site of heme copper oxidases.
The ROSA26 mouse mutant was originally produced by infection of embryonic stem (ES) cells with a ROSAβ-geo retrovirus [20].
A tah18 mutant was originally identified in the laboratory through a genetic screen for synthetic mutants with pol3-13, a thermosensitive allele of POL3 [1].
The stm-dgh6 mutant was originally identified in the Versailles T-DNA collection.
The vangl2/trilobite mutant was originally described as tri m209 (Jessen et al., 2002).
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These specific mutants were originally described elsewhere [ 2, 26].
Mutants were originally obtained in the Sippe 50 background and mapped by crossing with 165E.
ATPalpha DTS mutants were originally isolated based upon their temperature-dependent paralysis phenotype.
This effect is remarkable considering that these mutants were originally engineered for different emission colors with LH2 1.
det1 mutants were originally identified via their light-grown phenotype when grown in the dark (Chory et al. 1989).
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