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This result indicates that ral1 mutant was more resistant to Al than WT.
This result was consistent with the notion that ral1 mutant was more resistant to Al than WT.
A dose-response experiment revealed that ral1 mutant was more resistant to Al than wild-type (WT) at all Al concentrations tested.
Since ral1 mutant was more resistant to Cd as well, we want to know whether Cd accumulation pattern in the mutant was altered or not.
The results of limited proteolysis indicated that Glu138Pro mutant was more resistant against trypsinolysis and this variant was less quenched in both acrylamide and KI quenching experiments.
The double mutant was more easily dissociated than was the wild type.
The nfr1 mutant was more responsive to M. loti inoculation compared to nfr5 and nup133.
As expected, the microcystin-deficient mutant was more susceptible to 1 µM H2O2 (Fig. 7).
The pmt1 mutant was more susceptible to SDS medium than wild-type strains and the mutant cells were enlarged.
However, the hsp90.2-3 hsp90.2-3s mutantesistant than the control for a given concentration of Me-JA (Fig. 8).
However, the mutant was more sensitive to cell lysis caused by oxacillin, a cell-wall synthesis inhibitor.
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