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The proliferation level through the truncated mutant was even higher than that of non-transduced HSCs with the stimulation of a native cytokine, thrombopoietin.
The mutant was even further impaired in its competitive abilities against the parental strain after 7 days of growth, resulting in a growth ratio of 1∶0.04.
The hrk1Δ hog1Δ mutant was even more sensitive to fluconazole and ketoconazole than the wild-type strain, implying that Hrk1 is involved in controlling azole sensitivity in a Hog1-independent manner.
With increasing amounts of either compound, growth of the pop2-5 ssadh-2 mutant was even more affected than that of single ssadh-3 mutants grown on SSA, most likely due to a more severe phenotype of ssadh-2 than ssadh-3 mutants (Figure 6A; [26A).
Conversely, no restoration of YwbN secretion was observed in the tatAdCd mutant upon expression of a plasmid-borne tatCd gene alone, and YwbN secretion by the tatAdCd mutant was even completely suppressed by the presence of a plasmid-borne copy of tatCy.
As expected, the Leu294Gly mutant was even more Mg2+ sensitive than the Leu294Asp variant.
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The cytokine levels induced by this H1N1 mutant were even higher than those from H5-infected cells.
Remarkably, the double mutant is even more distant from the myb28 mutant than would be anticipated from the effect of myb29 alone.
However, these experiments demonstrate that the pmt1 mutant is even more attenuated for survival in vivo compared to pmt4 mutants, despite similar in vitro temperature- and cell wall-sensitive phenotypes.
However, transcription levels of espE in the eccCb1::tn mutant were even further decreased.
Under 100 mM NaCl treatment, growth inhibition of sos2 mutants was even more pronounced.
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