Sentence examples for mutant to both from inspiring English sources

As a final proof of concept, we hybridized the DNA from bulks of an F2 mapping population of a Sulfur and Selenium ionomics mutant to both the Arabidopsis ATTILE1R and ATSNPTILE arrays, which produced almost identical results.

In the case of Notch1, a decreased response of the loop-deletion mutant to both Jagged-2 and Delta-like-1 ligands is observed in co-culture reporter gene activation assays, as predicted by the reduced number of receptor molecules at the cell surface (Figure 7A,B).

Significantly differentially expressed genes were identified by comparing the results with the knockout mutant to both wild-type and complemented strain results by using a t test (at a P level of <0.05, with Benjamini and Hochberg multiple testing correction) and a change threshold of >2-fold.

Then, different binding affinities of the EGFR TK mutants to both adenosine triphosphate (ATP) and erlotinib could explain the heterogeneous kinetics of [11C]erlotinib in the two erlotinib-sensitive tumors.

Here, we use the site-specifically tethered chemical nucleases and functional characterization of directed T7 RNAP mutants to both reveal the architecture of the duplex DNA that flanks the transcription bubble in the T7 RNAP initiation and elongation complexes, and to define the function of the interactions made by these duplex elements.

We compared transcripts whose abundance increased among mutants to both the sRNAs and the AtSet3 list of sequences to identify direct miRNA targets.

The sensitivity of cysl-1 mutants to both RA/H2S and RA/HCN suggest that cysl-1 acts downstream of cysl-2, and cysl-1 mutants are being overwhelmed by the H2S produced by cysl-2 during HCN detoxification.

This explanation is now outlined in the appropriate section of "Are the Urzyme activities authentic?" point 5. Regarding (ii), we are in the process of testing active-site mutants to both 46-mers.

An additional E175A mutation allows this mutant to use both NAD+ and NADP, and this mutant that is most versatile for cofactor regeneration is termed 17X-PTDH.

Soucek et al. (2008) used a dominant-interfering Myc mutant to determine both the therapeutic impact and side effects of Myc inhibition in a preclinical mouse model of Ras (OMIM 190020 -induced lung adenocarcinoma.

The ability of most MSL1 mutants to bind both MSL3 and MOF was unexpected given the results with polytene chromosomes.