The phrase "mutant that was" is grammatically correct and can be used in written English.
It is commonly used in descriptive or explanatory sentences to introduce a characteristic or trait of a specific mutant. Example: The giant, green mutant that was terrorizing the city had sharp claws and glowing red eyes.
However, a deletion mutant that was not lacking an upstream region from the AOX2 promoter showed a response to oleic acid.
Based on modeling of the hydrophobic spine assembly, we also engineered a constitutively active mutant that was independent of Ras, dimerization, and activation-loop phosphorylation.
We developed an innovative method to achieve functional visualization of 5-HT4R dimers by immunofluorescence, taking advantage of the 5-HT4-RASSL (receptor activated solely by synthetic ligand) mutant that was engineered in the laboratory.
A recent study showed that an Δhfq mutant that was non-motile exhibited an invasion defect [20].
This is in contrast with our GFP-Dnmt1ΔCXXC mutant that was designed to retain the β-sheet structure.
As predicted, the lipin-1α mutant that was deficient in nuclear localization also showed a defect in stimulation of MEF2 transcription (Figs. 7B-7D).
In addition, a mutant that was previously shown to have a defect in RNA binding Npl3-1200) and a truncated form of Npl3 containing only the two RRMs (aa 121 280) were also tested [11], [12], [33].
We therefore tested if Praf2 overexpression could induce cell death, and if this could be blocked by the concomitant expression of Bcl-xL or the Bcl-xLΔTM mutant that was shown to be unable to bind to Praf2.
Single molecule mechanics using a laser trap, gave a unitary displacement and force for the mutant that was similar to wild-type, but the attachment time to actin following the unitary displacement was markedly reduced.
We identified an STM mutant that was able to grow in the lungs of SP-A−/− but not SP-A+/+ mice, as indicated by the presence of a PCR amplified oligo tag in bacterial output from SP-A−/− mouse lungs, and the absence of a PCR amplified oligo tag in bacterial output from SP-A+/+ mouse lungs (Figure 1A).
Using the GP42-SeV mutant that was previously described [6], the GP42-GFP vector was generated [19] and the HA gene of influenza PR/8 virus was introduced in the intergenic sequence of the viral genome between the M and the F genes to generate the GP42-H1 vector (Fig. 1A).
