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The Bmpr1a Bmpr1b mutant provides a model for a subclass of HPE in humans: midline inter-hemispheric HPE.
The genetically and phenotypically defined mutant provides a vehicle for investigating the important genes responsible for non-race-specific resistance to two distinct diseases.
The mutant provides a useful tool for investigating the important genes responsible for non-race specific resistance to two distinct diseases.
This GFP151TAG mutant provides a rapid qualitative assay of amber-suppression efficiency in E. coli; it has been demonstrated previously that at position Tyr151 introduction of unnatural amino acids does not interfere with the fluorophore or affect the correct folding of the protein [17].
Thus, the ldox mutant provides a system that has leucoanthocyanidin but not cyanidin.
Furthermore, the roxy1 mutant provides a most suitable system to determine the crucial functional domains of CC-type GRXs.
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Thus the Bdr mutant provides an important link between sensory behavioural abnormalities and a specific synaptic deficit caused by a single amino acid change.
The WDR5 F266A lncRNA-binding deficient mutant provides an ideal reagent to act as nonbinding control in the systematic genome-wide identification of functional WDR5-binding RNAs.
These suppressors also suppressed act1-1-related salt sensitivity and the defect in actin repolarization of the rvs161delta mutant, providing a link between sphingolipids and actin polarization.
The sctt mutant provided a unique opportunity to perform a structure-function analysis at single amino acid resolution of this evolutionarily conserved motif within an intact developing organism.
The use of this rhodopsin mutant provided a number of advantages.
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