Sentence examples for mutant plants indicate from inspiring English sources

The divergent sequence of CYTOSLEEPER, represented by the long branch-length in Figure 3, and the lack of an apparent phenotype in mutant plants indicate that this gene might be pseudogenized or has obtained a different function as DAYSLEEPER.

These proteins were absent in the ago2-1 mutant plants indicating that they represent isoforms of AGO2 due possibly to posttranslational modification or cleavage by proteolytic enzymes.

It is noteworthy that all identity changes observed in tomato plants overexpressing TAGL1 gene are coincident to those observed in Alq mutant plants, indicating that their phenotype is promoted by the ectopic expression of TAGL1.

Analysis of ida mutant plants indicates that IDA regulates floral organ abscission through an ethylene insensitive pathway.

Exogenous sulfide prevents ATG8 protein accumulation and lipidation in des1 mutant plants, indicating that intracellular H2S regulates autophagy.

In addition, miR5640 precursor accumulated in the DCL1 (dcl1- 9) mutant plants, indicating that miR5640 precursor is processed by DCL1 as most miRNA precursors [ 69].

No significant differences were found in Cd content between wild-type and mutant plants, indicating that the absence of induction of ethylene production in mutants was not attributable to a decreased Cd uptake (Table  1).

Interestingly, the biosynthesis gene DAD1 was strongly down-regulated in the root tips of pdr1 mutant plants, indicating a feedback regulation and cross-talk between SL biosynthesis and transport.

Surprisingly, we observed that a longer light period decreased the fertility of p5cs2 mutant plants, indicating that under these conditions P5CS1 is not sufficient to cover the demand for proline during reproductive development.

Mutations or chemicals that impair or block the transfer of electrons along the ETC to form ROS efficiently suppress the phenotypes of mod1 mutant plants, indicating that the ROS generated in mitochondria through ETC plays an essential role in triggering plant PCD.

This initial construct, termed minimal promoter (pBBmin), consists of 366 bp non-transcribed sequence and the entire 5' UTR and is sufficient to rescue the petal overgrowth phenotype of bb-1 mutant plants, indicating that it contains all necessary regulatory elements for BB expression regulation.