Sentence examples for mutant model in from inspiring English sources

Even under circumstances where low incorporation of these cells is observed, their importance has been supported by bone marrow transplantion studies using the Id1 mutant model in which the EPC function is completely impaired [5], [6], [10].

Note that we employ the same mutant model in [ 22] for comparison.

In the present study we used the high growth (hg) mouse mutant model in combination with chromosome 2 (MMU2) congenic and subcongenic strains to map genes that affect body composition, and genes that may interact with the Growth Hormone/IGF signaling pathways [ 5- 7].

In addition, studying the mutant models in their natural habitats, or modeling certain aspects of their life history in the laboratory, enables assessment of environmental influences on the symptoms and course of diseases.

In this study, we analyze skeletogenesis in two 'muscleless' mouse mutant models in which the formation of skeletal muscle development is disrupted; Myf5 nlacZ/nlacZ MyoD −/− and Pax3 Sp/Sp (Splotch).

Using a gene-targeting approach we generated two novel knock-in IGF1 mutant models, in which the native Igf1 gene was replaced by Des-IGF1 (KID) or R3-IGF1 (KIR).

On the other hand, z13 displays "B" duration and out-degree in ASTD L unlike in ASTD W. As shown in Additional file 4, per mRNA has a longer periodic oscillation in the mutant model than in the normal one.

Protein QC pathways have classically been elucidated using mutant model substrates in yeast (Jung et al. 1999; McClellan et al. 2005; Wolf and Schafer 2005).

The findings reveal that the maximal LE converge to positive real numbers thus providing evidence for chaotic dynamics of the wt and cwo-mutant models in both LD and DD conditions (Figures 3a and S5).

Furthermore, the position of ZT = 0 remains restricted to a very small neighborhoods on the limit cycles of the wt and cwo-mutant models in LD (see Figures 3b c arrows).

Our results are instead consistent with the second mechanism, as H3-L61X modeled (modeled in Figure 6B) that are predicted to disrupt the H3-L61 microenvironmenthroughgh the introduction of polarity/charge or bulk or by interfering with H3 polypeptide flexibility all cause strong perturbations in Spt16−gene interactions.