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Higher EDS1 and NPR1 activity at 12 hai in the mutant may indicate a higher level of SA signaling that is unfavorable to the needed JA signaling.
This exaggerated sensitivity of the double knockout mutant may indicate that Asc1 and Pkc1 proteins act through different mechanisms to maintain the integrity of the cell-wall.
The strong brown pigmentation in the 7Y mutant may indicate misregulation of plant defenses.
These matter-containing vacuoles are most likely autolysosomes, and their absence in the mutant may indicate a defect in autophagy.
The Unc phenotype of the mnm-6 overextension mutant may indicate that the gene functions in guiding other neurons.
The combined effect of the double mutant may indicate a combination of effects associated with loss of both histone H3K9 and H3K14 acetylation.
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A combination of this result with fast learning in young mutants may indicate that they might be able to re-learn faster than the older mutants or young wild-type zebrafish.
The vectorial variations in the mutants may indicate a decreased tubule stability and vulnerability to distortion.
Furthermore, the phenotype of Δ gpr-25 mutants may indicate a role for actin polymerization in this process.
The observed geographical structure of FCA variation and its correlation with morphologies expected from Arabidopsis mutant studies may indicate that developmental pleiotropy played a role in the diversification of the mints.
Assuming a pure tumor sample, this intermediate level of mutant allele may indicate intratumoral heterogeneity for LOH, with an admixture of tumor cells with and without LOH.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com