Sentence examples for mutant lines deficient from inspiring English sources

To explore other mutants showing phenotypes similar to the dPob null mutant, we examined a collection of 233 mutant lines deficient in Rh1 accumulation in photoreceptor rhabdomeres obtained in an ongoing ethyl methanesulfonate (EMS) mutagenesis screening.

Although the family I TRFL proteins are capable of binding telomeric DNA in vitro, examination of Arabidopsis mutant lines deficient for either TRFL1, 2 or 4 did not reveal any phenotype expected to be associated with altered telomere metabolism [59].

To explorer other mutants showing phenotypes similar to dPob null mutant, we examined a collection of 233 mutant lines deficient of Rh1 accumulation in photoreceptor rhabdomeres, obtained in an ongoing ethyl methanesulfonate (EMS) mutagenesis screening.

For this, we used barley mutant lines deficient in Ror1 and Ror2 (required for mlo-specified resistance) [ 19, 20], Rar1 (required for Mla12 resistance) [ 21] and Rom1 (restoration of Mla12-specified resistance) [ 22].

To confirm that ss1, be1, and be2b single mutant and double mutant lines were deficient in the relevant proteins and enzyme activities, we conducted zymogram analyses by semi-quantitative native- polyacrylamide gel electrophoresis (PAGE) and immunoblotting assays of the soluble protein fraction (SP) from developing endosperm at 12 days after flowering (DAF).

DM molecules are critical for the classical MHC class II function, as exemplified by the observation that human mutant cell lines deficient in DM molecules, and also antigen-presenting cells from the DM-knockout mice, exhibited failure in proper MHC class II peptide presentation [ 7- 10].

Mutant hamster cell lines deficient in NHEJ (XR-C1, XR-V15B) and HR (CL-V4B, V-C8) with their parental cell lines (CHO9, V79B and V79) were used.

Because 21 nt ta-siRNAs are produced by DCL4 [21] [23], this result confirms that these dcl4 mutant lines are highly deficient for DCL4 activity.

It is remarkable that these general alterations are observed even in mutant lines that are deficient in steps early or late in the desaturation pathways.

Mutant and antisense lines deficient in catalase exhibit chlorophyll bleaching and necrosis in the interveinal areas, a phenotype that can be rescued by elevated CO2 or low light [ 44, 45].

We tested four different cerebellar mouse mutant lines, which suffer from deficient processing in their Purkinje cells, molecular layer interneurons or granule cells, on the Erasmus Ladder to study their basic walking patterns, locomotion adaptation to perturbations and interlimb coordination.