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Each independent gene mutant is identified as X and Y.
In this study, a carbon and light insensitive (cli186) mutant is identified and its molecular defects characterized on a genome-wide scale, using a multinetwork approach to identify the genes, biological processes and regulatory/metabolic networks affected in the cli186 mutant.
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A mutant was identified that was 132 times more specific for ABTS.
Each CS-HA1 mutant was identified by Western blot as a single polypeptide of the predicted molecular weight.
The color of each isolate or mutant was identified with the PANTONE Color Standard book [Eiseman, L., Herbert, L., 1990.
The double homozygous mutant was identified by PCR; this mutant was then crossed to homozygous ape1L-1.
The enz mutant was identified based on its altered pigment pattern.
This mutant was identified in a forward genetic screen for essential genes [37].
The third additional peptide which phosphorylation was not changed in the CDK9 S175 mutant was identified as C-terminal 346GSQITQQSTNQSR358 peptide.
The mutant was identified as weakly resistant to levamisole due to significantly reduced postsynaptic density of AChRs [48].
The pgr7 mutant was identified from fast neutron mutated M2 seeds (Ler) obtained from Lehle Seeds (Round Rock, TX).
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