Sentence examples for mutant in addition from inspiring English sources

Up to 38 different structures of Nod factors were detected, being higher under salt stress, except for the nodD5 mutant; in addition, a high number of structures was synthesized by the nodD4 mutant in the absence of any inducer.

For imatinib, bosutinib, and nilotinib, there was one highly resistant mutant in addition to the known T315I mutant (V299L for bosutinib and E255V for nilotinib), whereas T315I was the only mutant with a high relative resistance against dasatinib [46].

We also provide data on the loss of Jen1 degradation in the ypt6Δ mutant, in addition to that in the vps52Δ mutant already present in the original version of the manuscript (new Figure 4F).

Because we have assumed that the ancestral states of SNPs along the chromosomes are known, for every haplotype, we can determine the break points of the ancestral haplotype caused by the recombination events nearest to the mutant, in addition to the locations where mutations occurred within each ancestral haplotype region.

Instead, the E545K mutant specifically bound activated Cdc42 in vitro and microinjection of E545K was associated with the formation of cellular protrusions, providing some preliminary evidence that changes in protein protein interactions may play a role in the oncogenicity of the E545K mutant in addition to the well-known changes in lipid kinase activity.

Several LPS mutants in addition to lpcC, manB, manC and kdtA were identified and studied in this work.

The asdA gene from E. ictaluri complemented S. enterica ΔasdA mutants, in addition the Asd enzymes from E. ictaluri and S. enterica share 81% similarity.

Thus, physiological analysis of these and other mtSSB mutants, in addition to development of new in vitro assays that reconstitute fully the mtDNA replisome, will be informative in understanding the mechanism of mtDNA replication.

It will also help to reveal the larger picture of the cry1 signaling pathway by examining the protein dynamics of other factors in the blue light pathway in cry1 mutants, in addition to cry1 itself.

Then, we have performed invasion assay and epithelial-mesenchymal transition marker study in human bronchial epithelial cells overexpressing AQP5 versus its mutants in addition to mock control, followed by further molecular interaction studies to elucidate AQP5 mediated pathway.

Because lin-17/Fz mutants show some underextension defects similar to mig-5/Dsh and bar-1/β-catenin mutants, in addition to their overextension phenotype, we propose that Fz/LIN-17 activates both a canonical pathway to stabilize β-catenin and prevent axon termination, and an uncharacterized pathway that triggers axon termination.