Sentence examples for mutant embryos exposed from inspiring English sources

Since the cleft palate phenotype is fully penetrant in Viaat and Gad1 homozygous fetuses and newborns, the rescue of the cleft palate phenotype in mutant embryos exposed to muscimol is quite clear.

The cuticle preparations were visualized by DIC optics using a 10x 0.3 NA objective on a Leica DM IRE2 inverted microscope and a cooled ORCA ERG 1394 CCD camera (12-bit, 1344×1024 resolution, Hamamatsu Photonics, K.K .. We thank Joao Marques for qPCR confirming absence of increased viral load in the mutant embryos exposed to the T-step relative to controls.

To determine if dcr-2 mutant embryos exposed to a temperature step could nevertheless correct for the early abnormalities in segmentation, exoskeletal cuticles were prepared from larvae that had developed in a temperature step for the first 200 minutes of embryogenesis, and then further developed at a uniform temperature of 24°C.

In fact, when a greater difference in the rate of development is induced in WT embryos by placing them in a temperature step with a larger difference in temperature (anterior at 27°C and posterior at 17°C), a larval phenotype was observed which is similar to that of the dcr-2 mutant embryos exposed to a temperature step with a smaller difference in temperature.

Untreated mib mutants lacked 5HT neurons but, remarkably, 5HT neurons were generated in mib mutant embryos exposed to VPA (Fig. 5C).

In addition, when Fancd2 −/− Aldh2 −/− mutant embryos were exposed to ethanol in utero, most developed severe physical abnormalities and died.

Although only 7% of WT embryos exposed to low dose ATRA showed overt HPE or NTDs, 100% of Twsg1−/− mutants manifested HPE compared to 17% without exposure to ATRA (Table 1; Fig. 1; P=6×10−12).

Although only 7% of wild-type embryos exposed to RA showed overt HPE or neural tube defects (NTDs), 100% of Twsg1 −/− mutants exposed to RA manifested severe HPE compared to 17% without RA.

Relative levels of gene expression were compared among control embryos and embryos exposed to these constructs during development.

As shown in Figure 7D, the hatching rate of embryos exposed to the mixture groups at BPA concentrations of 5 mg/L was significantly retarded compared to that of embryos exposed to the BPA alone-exposed groups.

We found that fgaB but not fgaA mRNA is induced in fly embryos exposed to hypoxia (5% O2), and that this induction is Sima-dependent, as it was completely blocked in sima mutant embryos (Fig. 2B).