Sentence examples for mutant embryos also from inspiring English sources

A small fraction (1.7%) of the mutant embryos also displayed micro- and macronuclei as well as irregular arrangements of nuclei (Figure 4B).

Like γCOP, sar1 mutant embryos also show a reduced secretion in the SG (Figure 3G), suggesting both COPI and COPII are required for efficient luminal deposition in the SG.

In addition to these patterning defects, Hexdact/+ mutant embryos also exhibited reduced expression of both Fgf8 and Snail in the anterior portion of the primitive streak (Fig.4K L).

These homozygous mutant embryos also show cardiovascular and heart defects, hydrops fetalis, and bone defects [4,7,8].

Mutant embryos also fail to form limb buds completely, indicating that FGFR2 Ig domain III is essential for limb initiation.

The relative decrease in Six3 expression in bkr versus gnz mutant embryos also correlates with the extent of anterior truncation.

Mutant embryos were also reduced in size compared to heterozygous littermates suggesting a defect in epiblast proliferation.

Moreover, the expression of the Drosophila FGF receptor Htl [58], which is downregulated in wgCX4 mutant embryos, was also rescued in wgCX4; cnomis1 double mutants (Fig. 7G and 7H) [55].

While embryonic patterning was rather normal in γCOP mutant embryos (see also below), they were smaller than wild type embryos and displayed weakly pigmented cuticles with poorly differentiated denticles (Figure 3); some of the mutants also displayed a partial dorsal open phenotype.

mRNA stabilization throughout aub mutant embryos was also validated using in situ hybridization experiments for five mRNAs.

Both modSP 1 and psh 1 ; modSP 1 double mutant embryos were also less fit compared to wild-type embryos after injection with Aspergillus oryzae proteases.