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Consequently, the mutant demonstrates a loss of energy transfer efficiency.
As DET1 is a part of the CCA1/LHY repressor complex, the det1 mutant demonstrates a short period phenotype similar to that of cca1 or lhy mutants.
The oga-1 ok1207) oga-1 ok1207strates a substantial increase in O-GlcNAc-mutantedemonstratesvels asubstantiald by increaseot of whole aninal lysate, while the O-GlcNAc-modifiedant has only residual staining.
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Analysis of this mutant demonstrates an accelerated onset of touch neuron defects.
The ODC_N125K/M140K double mutant demonstrated a significant inhibition by AZ, and the IC50 value of this mutant was 0.08 µM, three-fold smaller than that of ODC_WT.
The K14A mutant, demonstrated a 50% increase in the Km for ATP with a Km value of 86.7 µM as against 56.8 µM for the WT (Table 2).
cerevisiae mutant demonstrated a functional urea transport.
Both phosphorylated SMAD2 and p21 levels were lowered in the homozygous mutant, demonstrating a suppression of the TGFβ pathway.
In addition, the crzAΔ scrC3 mutant demonstrated a far greater level of pmcA mRNA accumulation than the wild type strain.
In each of the four combinations, the double mutant demonstrated a more severe root growth defect than either of the single mutants.
If such a mutant demonstrated a compromised SAR response, it would greatly strengthen the claim that AtPNP-A is indeed involved in the SAR response pathway.
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