Sentence examples for mutant data of from inspiring English sources

Using mutant data of the N-CDA, we mapped critical residues for packaging of APOBEC3G into viral particles on the new structure model of the huAPOBEC3G N-CDA.

We show here that the BMP-receptor interaction meets the conditions for a Schnakenberg-type Turing pattern, and that the resulting model reproduces available wildtype and mutant data on the expression patterns of BMP, its receptor, and Fgfs in the apical ectodermal ridge (AER) when solved on a realistic 2D domain that we extracted from limb bud images of E11.5 mouse embryos.

We combined the pif-mutant data of Khanna et al. [28], Al-Sady and colleagues [46] as well as Leivar et al. [29] with our over-expressing lines phyB-GFP-1 to 4 relative to the phyB level of Col WT, since all experiments were conducted under saturating red light conditions for four days.

Some of our phenotypic variability may stem from inconsistent shRNA knockdown (Mohr and Perrimon, 2012), but the mutant data suggest much of the variability must emerge from the network response to bcd depletion (Fig. 1C).

The set of 520 genes that we found to be differentially expressed during wild type nodule formation (wild type temporal profile data set) was used as a reference set for the comparison of the transcriptomes of the Fix− nodules of the different mutants (mutant data set) and those of wild type nodules in the Jemalong J5 background induced by S. meliloti Sm1021 or Sm2011.

We generated 24 time series, including wildtype and knock-out mutant data, with a total of 202 time points (≪1 % of the 2 possible states in the system).

The absence of doubly homozygous mutant data precludes a formal assessment of epistasis.

Part of the work related to the assembly of mutant data set was done by CD when he was an undergraduate student.

Statistical significance for both red and brown pigments was determined by using an ANOVA followed by Student's t-test with Bonferroni-corrected P values between the mean of the experimental dCTCF mutant data sets and the mean of the results from the appropriate control cross.

Thus, the double mutant data confirms the position of DRH-1 as an upstream factor essential for the generation of robust levels of antiviral siRNA in response to infection.

An interesting observation is the lack of requirement for Slit ligand in the luciferase experiments, whereas the Slit2 and Slit3 mutant data indicate the requirement of Slit-Robo binding.