Sentence examples for mutant cells after from inspiring English sources

(A ) Metabolic profiling of wild type and CtpSE277R mutant cells after addition of cytidine to minimal media.

The defective actin dynamics and cell polarization observed in rad2C65Δ mutant cells after UV irradiation suggest that Rad2p plays a role in cell polarity regulation.

Measurement of LC3-II/LC3-I ratimmunoblotinging confirmed robust autophagy induction in both control and mutant cells after starvation (Fig.  4B).

As expected, this proportion increased substantially upon selection, reaching a maximum of 82% mutant cells after selection for a further 7 days in puromycin (Fig. 4B,C; supplementary material Fig. S1).

To demonstrate that autophagy is induced during fermentation, we performed electron microscopy (EM) analysis with pep4 homozygous mutant cells after two days of fermentation compared with log phase pep4 homozygous mutant cells.

The arrested cell division and decreased lifespan of rad2C65Δ mutant cells after UV exposure suggest that the C-terminally truncated XPG protein in XPG/CS cells causes cell cycle arrest and decreased cell longevity, and this resulted in the absence of increased cancer incidence in the presence of DNA damage in CS patients.

Further selection resulted in a loss of AGO1 mutant cells, and after 12 days of selection no mutant cells were visible.

Other hyphal-associated genes, which were similarly expressed in wild type and mutant cells 1 h after infection, were down-regulated in the mutant at later time points, for example ALS3, HWP1 and SOD5 (Fig. 3 C).

We investigated engulfed C. glabrata cta1Δ, pex3Δ, atg11Δ, atg17Δ, pex3Δ atg17Δ, pex3Δ and11Δ atg11Δg11Δ atg17Δ mutant cells expressing GFP CgCta1 after 5 and 24 h (Fig. 6A and E).

The amount of fragmented DNA decreased 35% after 3 hr and 45% after 6 hr in the wt cells, which compared to a maximum of 27% reduction observed in mutant cells 6 hr after DNA damage.

In contrast, mutant cells irradiated 33h after 4-OHT treatment (n = 346) were able to re-enter mitosis after a shorter period of time indicating that cell cycle arrest in response to DNA damage was reduced in Omcg1-deficient ES cells.