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Alternatively, an auxotrophic mutant can also be utilized in which specific nutrient withdrawal will result in inhibition of the bacterial growth.

The posterior wing crossvein in the mutant can also project ectopic longitudinal veins.

This mutant can also be viewed as an H18R mutant of pramlintide.

S. Enteritidis SPI2 mutant can also induce significant increase of antibodies in chickens [ 9].

Furthermore, ectopic expression of wild-type SIRT5 but not a catalytic defective mutant can also restore H2O2-induced cell death in SIRT1 knockdown cells.

Whether the mutant can also suppress pre-existing tumors is unclear, and this important question is readily addressable by expressing the mutant protein in P53 cKO mice.

Considering that wild type (WT) TET1 can positively regulate the srGAP3 expression, we wonder whether the TET1 mutants can also modulate the srGAP3 expression.

Truncated nonsense mutants can also arise from insertion or deletion mutations producing premature stop codons.

Seeds from these Syngenta (Research Triangle Park) mutants can also be obtained from ABRC.

These mutants can also help clarify patterns of evolution between VS and UVS visual systems in birds.

While avoiding the disadvantages associated with the use of inhibitors, the use of suppressor mutants can also present its own problems.