Sentence examples for mutant background indicating from inspiring English sources

Exact(6)

By contrast, all constructs revealed GUS-positive cells in the cpc try mutant background indicating that this regulation event involves the trichome patterning activators.

The effect of ΔJpx/+ was rescued by placing it into a Tsix mutant background indicating the opposite action of both genes (Tian et al. 2010).

GFP expression in the histoblast nests did not increase in the miRNA mutant background, indicating indirect regulation of EcR by miR-965.

Even the low copy insertions showed stronger expression in the pqe-1 (pe334 ) mutant background, indicating that the effect of PQE-1 also covers low copy transgenes produced by MosSCI.

We previously showed that mma-1 RNAi) mma-1 RNAitheticausesyonic lethality and synthetic midlarval arrembryonice fzo-1(tm1133) lethalityunction mutand background, indicating that mitochondrial hyperfusyntheticssential for viability in response to midlarvali) [ 11].

Neither NFR construct spontaneously induced nodule organogenesis in a symrk-3 (null mutant) or symrk-10 (kinase dead mutant) background, indicating that the formation of nodules is depended on the presence of kinase-active SYMRK.

Similar(54)

For most tests of gland morphology, we either crossed ivIs12 into the mutant background to label glands or generated new extrachromosomal transgenes by injecting the phat-1:: YFP reporter into the desired mutant background (indicated as "Ex" in Table 1).

By contrast, flies homozygous for the vg PRE showed a loss of silencing at all three sites in the Pc XL5 mutant background, indicated by an increase in eye pigmentation.

This is in line with the observation that dominant active variants of CCaMK were able to restore nodulation and infection in symrk mutant backgrounds, indicating that a main function of SYMRK in symbiosis is the activation of CCaMK (Hayashi et al., 2010; Madsen et al., 2010).

Reporter gene fusions to the groESL and dnaKJ operons transformed into these mutant backgrounds indicated that sig1 is required for the heat shock induction of groESL and dnaKJ, whereas sig2 mutants show a more moderate defect in dnaKJ induction and are not impaired for groESL induction.

Interestingly, though, HRK1 can be strongly induced in response to oxidative and osmotic stress in the ssk1Δ mutant background, strongly indicating that unknown signaling component(s), other than Ssk1, can activate Hog1 and subsequently induce expression of Hrk1.

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