Sentence examples for mutant background demonstrating from inspiring English sources

This defect can be rescued by ubiquitous γCOP expression from a tubulin1α-promoter in the mutant background demonstrating that γCOP is required for epidermal morphogenesis (Figure S1).

Moreover, the overexpression of Sak could not drive the formation of these structures in a DSas-4 mutant background, demonstrating that DSas-4 is required for their formation (not shown).

We also investigated HDR efficiency in a mutant background previously demonstrated to bias DNA repair toward HDR.

Thus, the nonphosphorylatable mutants frequently disrupt anterior development in both osk mutant and wild-type backgrounds, demonstrating that Slimb-dependent degradation of Osk is important for normal anterior-posterior axis formation.

Early literature indicated that the Li 1 mutant in a TM-1 genetic background demonstrated a reduced rate of crystalline cellulose deposition during primary cell wall synthesis and an increased rate during SCW synthesis, resulting in a "thickened" appearance of the cell wall in the mutant [ 18, 30].

Using transgenic fish with a heat-shock inducible tbx6 gene in the fss/tbx6 mutant background, we further demonstrate that ubiquitous fss/tbx6 expression has spatially distinct effects on recovery of the dermomyotome and segment boundaries, suggesting that the mechanism of Fss/Tbx6 action is distinct with respect to dermomyotome development and segmentation.

In our original description of pxy, the pxy-1 allele in L er background demonstrated a stronger organisation defect than mutants in the Col background (Fisher and Turner, 2007) (supplementary material Fig. S6).

In addition, hemocyte-specific expression of wild-type SCAR completely rescued developmental migration defects in a SCAR Δ37 mutant background (SCAR rescue), further demonstrating an autonomous requirement for SCAR.

Genetic dissection of this trait, through QTL mapping and characterization of introgression lines carrying the alternative low- and high-density alleles in a mutant background, identified three candidate genes with demonstrated or predicted roles in trichome development: ENHANCER OF TRY AND CPC2 (ETRICHOMELESS1ELESS1 (TCL1), and TCL2 (Hischer et al. 2009).

Expression of wild-type SCAR specifically in hemocytes in a SCAR Δ37 mutant background (SCAR rescue) significantly rescued hemocyte speed, demonstrating a cell autonomous requirement of SCAR for effective migration.

As expected, overexpression of CVY1 in cvy1 mutant background was sufficient to rescue the cvy1 phenotype, demonstrating that CVY1 gene knockout is indeed responsible for the phenotypic characterization in cvy1 mutant phenotype, and thus providing further confirmation of the correct genetic characterization of CURVY1 as a new " DISTORTED" gene.