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Six of the cell lines (NOMO-1, HEL, HL-60, SKM-1, THP-1, and TF-1) are TP53 mutant and three of the cell lines (EOL-1, MOLM-13, and MV4-11) are TP53 wild-type.
c RT-PCR of REL2 in rel2 mutant and three independent complementary lines c-rel2-1, c-rel2-2 and c-rel2-3.
Blots were run in triplicate and at least three mutant and three control samples were on each blot.
To distinguish these two possibilities, we measured conditioned place aversion induced by mild aversive signals in the mutant and three groups of control mice.
We have reproduced these results with independent RNAse protection assays (two experiments for the effect of each mutant and three experiments for the maternal effect).
We tested samples from the five mutant and five wild type control mice at young (13 weeks) age and three mutant and three wild type control mice at old age (20 months).
Similar(51)
Complete concordance for factor V Leiden genotypes was observed between the two methods in the 205-sample cohort comprising 139 wild-type, 62 heterozygous mutant, and four homozygous mutant individuals.
a Col-0, 129B08/nced3 mutant, and two N4C transgenic lines after 9 days of withholding water.
b Col-0, 129B08/nced3 mutant, and two N4C transgenic lines after 13 days of withholding water.
Fig. 2 Phenotypic comparison of 35-day-old, soil-grown Col-0, 129B08/nced3 mutant, and two OsNCED4-overexpressing transgenic lines.
Fig. 1 RT-PCR analysis of native AtNCED3 and transgenic OsNCED4 expression in Arabidopsis plants; Col-0, 129B08/nced3 mutant, and two OsNCED4-overexpressing (N4C) transgenic lines.
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