Our data identified the presence of pericranial muscle tenderness as predictor of response to onabotulinum toxin A in patients with complicated form of migraine such as MOH, the presence of pericranial muscle tenderness and support it as prophylactic treatment in these patients.
Consecutively 141 acute WLP and 40 ankle injured recruited from emergency units were examined after 1 week, 1, 3, 6, 12 months obtaining neck/head VAS score, number-of-non-painful complaints, epidemiological, social, psychological data and neurological examination, active neck mobility, and furthermore muscle tenderness and pain response, strength and duration of neck muscles.
Although myositis can cause muscle aches and muscle tenderness, weakness is usually the dominant symptom.
Symptoms include severe muscle stiffness (especially after sleep), malaise, weight loss, muscle tenderness, anemia, and fever.
The contribution of pelvic floor muscle tenderness to chronic pelvic pain (CPP) is well established in the literature.
Myofascial trigger points (TrPs) have been clinically described as discrete areas of muscle tenderness presenting in taut bands of skeletal muscle.
Pericranial muscle tenderness was assessed by a total tenderness score (TTS) involving 8 pairs of muscles and tendon insertions.
Persons with CTTH have an increased pericranial muscle tenderness.
A modified version of a previously published pericranial muscle tenderness score system was used to determine the pericranial muscle tenderness [16, 17].
We used a population-based reference population for comparison of pericranial muscle tenderness.
