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We aimed to evaluate the impact of gradual blood reperfusion on ischaemia reperfusion injury and to explain the pathophysiology of reperfusion injury in a rat cremaster muscle microcirculation model.
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The mouse cremaster muscle microcirculation is therefore a useful model to investigate the mechanism of leucocyte-endothelium interactions in tumour biology.
Based on these observations, we tested the hypothesis that rHuEPO given as a single dose of 400 U/kg would improve skeletal muscle microcirculation and tissue bioenergetics and thus ameliorate tissue metabolic dysfunction in a mouse model of severe sepsis.
NMES acutely affects skeletal muscle microcirculation; such effects could mediate the favorable outcomes.
A single NMES session affected local and systemic skeletal muscle microcirculation.
There is one study that showed systemic effects of a single NMES session on skeletal muscle microcirculation [16].
This study aimed to compare the effects on muscle microcirculation of a single NMES session using medium and high frequency currents.
The aim of the study was to establish a model of tumour microcirculation in vivo using the murine renal cell carcinoma cell line (RENCA) implanted into the mouse cremaster muscle, and subsequently to investigate the trafficking of syngeneic lymphocyte subpopulations into both the RENCA tumour and the surrounding normal cremaster muscle microcirculation.
We investigated skeletal muscle microcirculation in SSc patients using skeletal muscle BOLD MRI of the calf.
We hypothesized that PAH patients also have systemic muscle microcirculation alterations compared to healthy subjects.
Wide field, multichannel intravital microscopy of the cremaster muscle microcirculation was performed as previously described.
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CEO of Professional Science Editing for Scientists @ prosciediting.com