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Therefore, each muscle is able to influence all six degrees of freedom which makes the system kinematically and mechanically indeterminate.
The results explain how a single muscle is able to control the different movement parameters required for the proper function of ant mandibles.
Through the analysis of the actuation mechanism, it is shown that the sleeve muscle is able to provide a consistent increase of force capacity over the entire range of motion.
Normal skeletal muscle is able to efficiently repair itself in response to injury.
In view of the parallel phenotypes between mutant SOD1 and MCK-UCP1 mice, one can suggest that mutant SOD1 expression in skeletal muscle is able to trigger axonal degeneration through the direct implication of mitochondria.
Taken together, these data suggest that tumor cells are able to cross the vessel barrier and that skeletal muscle is able to "recruit" melanoma cells into the myogenic program and exert an inhibitory effect on tumor growth and invasion in vivo.
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After ten minutes stimulation, cells in this muscle were able to generate Ca2+ transients with every second stimuli.
Transgenic expression of Slit2 specifically in the muscle was able to diminish presynaptic deficits by Ctnnb1 mutation in mice.
Again, MUSCLE was able to discover an improved set of parameters that provided a faster (18.5%) and much more sensitive (104%) analysis (Supplementary Table S6).
Upon differentiation satellite cells isolated from soleus muscle were able to fuse and form multinucleated myotubes that displayed spontaneous contractions already after 48 h of differentiation (Movie 1 in Supplementary Material available online at http://dx.doi.org/10.1155/2015/594751).org/10.1155/2015/594751
Third, transgenic mice expressing Slit2 specifically in the muscle were able to diminish presynaptic deficits by Ctnnb1 mutation, including poor innervation, reduced vesicle density and active zone, and depressed mEPP.
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