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Impaired skeletal muscle function has been reported in patients with chronic obstructive disease (COPD), but such impairment is not homogenous and its distribution between the upper and the lower limbs is still unclear.
The increased production of free radicals and oxidative stress in the context of physical exercise, sport performance, and muscle function has largely been explored [1, 3, 5, 9, 10].
Cementless THR with a subtrochanteric femoral shortening osteotomy, rather than a proximal shortening osteotomy with distal advancement of the great trochanter to restore abductor muscle function, has been described as it avoids the risk of fibrous non-union of the great trochanter [7].
Whether 1,25 vitamin D is also effective to improve muscle function has been insufficiently investigated.
Traditionally, the main means of assessing muscle function has almost exclusively been through volitional testing [ 11].
Impaired skeletal muscle function has important clinical outcome implications for survivors of critical illness.
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Therefore, younger patients with better muscle function have more muscle mass with a higher production of cytokines.
Results from the case-control and RCT study on individual pain perception and muscle function have previously been published.
Patients with better muscle function had the highest concentration of CK, IL-1, and TNF- α compared with less muscle function.
Methods for measuring trunk muscle function have previously been established for younger adults [ 11, 12], but very little is known about these methods in the older adult population.
From the proteomics data, we found that proteins important in muscle function have several AGE modifications that are reduced on RIF treatment.
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