Sentence examples for muscle fiber density from inspiring English sources

Muscle fiber density did not differ among calf groups, but fiber diameter was greater in low than high and in crossbred than purebred offspring.

The least square analysis showed that there was significant difference (P<0.05) in muscle fiber density and some carcass traits among genotypes and that the breast muscle fiber density (BFD) of birds of A1A1 genotype was significantly higher (P<0.05) than that of birds of A2A2 genotype.

However, muscle fiber density was still reduced to 85.7%, 80.3%, and 73.8% of control for the 0.8, 2, and 5 ppm embryonic-only exposure groups, respectively, even after 16 weeks of development.

The aim of this study was to evaluate the effect of herbage allowance treatment of native pastures from calf conception to weaning on muscle fiber density and diameter and gene expression (insulin-like growth factor (IGF) system and adipogenesis) during the first year of age of purebred (Hereford and Angus) and crossbred (F1) dams offspring.

Muscle fiber density (FD) was determined to be 199 fibers/mm2 tissue based on a reported capillary density (CD) of 231 capillaries/mm2 tissue and a capillary-to-fiber ratio (CF) of 1.16 [73].

Due to the small sample size, we did not conduct a statistical analysis of muscle fiber mitochondrial density.

Based on our previous studies, we found a significant shift in muscle fiber type, mitochondrial density, and aerobic efficiency as seals switch from being a non-diving pup to a deep long duration diver as an adult.

Muscle atrophy is the loss of muscle mass resulting from a reduction in muscle fiber area or density due to a decrease in protein synthesis and an increase in muscle protein breakdown; these processes activate two major systems: the proteasomal (ubiquitin-proteasome system or UPS) and the autophagic-lysosomal system [ 1– 3].

ATR at the highest concentration (1 mg/L) interfered in the normal development of sarcomere muscle fiber seriously; the density of juvenile sarcomere was lower than the control group's significantly at 2 dpf, and the muscle fibers were arranged loosely).

This notion is supported by previous studies that have showed age-dependent muscle degeneration, loss of fiber density and vacuolization in of the indirect muscle fly [40] and increases in areas devoid of myofibrils in dmDys mutants fly hearts [80].

Four days after the initiation of injury, injection of adenoviral Ang-1 into injured muscles resulted in significant increases in in situ TA muscle contractility, muscle fiber regeneration, and capillary density.