Congenital myopathies cause weakness and poor muscle development in the early years of life, but generally they are not progressive.
From a nutritional point of view, it would be interesting to investigate genes with a crucial meaning for the muscle development in birds.
The objective of this experiment was to investigate the mechanisms through which different levels of dietary energy affect postnatal skeletal muscle development in ewe lambs.
In this report we characterize the novel SMA/Mstn model and demonstrate that Mstn inactivation does not significantly enhance muscle development in neonatal animals, nor does it result in an amelioration of the SMA phenotype.
Here we show that ladybird genes control leg muscle development in the fruit fly.
The four myogenic basic helix-loop-helix regulatory factors, MyoD, Myf5, myogenin, and MRF4, are crucial for muscle development in the embryo.
QSulf-1, the first member of the Sulf family to be described, was discovered in an analysis of muscle development in the quail embryo [20].
These results suggest that members of the Smyd family play conserved roles in muscle development in both vertebrate and invertebrate species.
Larval muscle development in Drosophila leads to the formation of ∼30 distinct muscle fibers arranged in a stereotyped pattern within each embryonic trunk hemisegment (reviewed in [1]).
It is required for smooth muscle development in the lungs helps in airway wall "modeling", and proper functioning of lungs throughout life [146], [147].
Given the clear defects in the larval musculature of ΔC3GMS animals, we investigated whether any of the molecules known to be important for muscle development in Drosophila was affected in the mutants.
