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Remarkably, nearly all the genes affected by Clo in the enriched muscle component and energy pathways were down-regulated, suggesting functional suppression of energy metabolism and motility.
These firm, circumscribed masses are known to possess a smooth muscle component, and may often have a significant extracellular matrix (ECM).
GO analysis showed that biological processes related to muscle development were mainly enriched in different stages for both breeds; these genes were associated with GO terms including myofibril, sarcomere, and myosin complex, which are involved in muscle component and structure.
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The device was created using mice cells, which were separated out into motor neurons and muscle components and fused into those parts.
There has been, however, some concentration of its proximal and distal muscle components and reduction in mass and efficiency of its middle components.
Successful skeletal muscle development relies upon the correct cyto-architectural arrangement of the various muscle components and abnormal arrangements can result in devastating effects on muscle function.
Muscle components and organization were the most significant biological processes, such as Z disc (P 0 7.6E-04), myofibril (P = 9.1E-04), and contractile fibre part (P = 9.8E-04 9.8E-04
A few years later, Panjabi[ 2] developed a model of segmental spinal stability where the potential roles of passive articular structures, active muscle components and neuromuscular control were exposed.
Because the diverse disease models used for these microarray experiments (including diabetes, cancer cachexia, chronic renal failure, fasting and denervation) have muscle atrophy in common, the commonly up- or downregulated genes are believed to regulate the loss of muscle components and are called atrophy-related genes or 'atrogenes' (Sacheck et al., 2007).
Studies suggest that flight or fight is the sum total of distinct components activated concurrently: a somatomotor (skeletal muscle) component, a visceromotor (autonomic) component, and a pain-modulation component (see Figures 1 and 2).
Atrophy may lead to an increased signal intensity of the muscle component on both T1- and T2-weighted MRI.
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