Sentence examples for muscle cell surface from inspiring English sources

The same enzyme detects the energy imbalance in a muscle cell that exercises where its activation also leads to the insertion of Glut4 onto the muscle cell surface.

To gain insight into the molecular nature of the muscle cell surface projections we employed additional fluorescent protein fusions that demarcate components of the cytoskeleton.

To test whether muscle cell surface projections were affected when muscle elongation was perturbed, embryos were electroporated with sna>Bix and Tbx6b>PH-YFP (Figure 6M P and Movie S10).

Muscle cell surface projections were most abundant at the neurula stage (Figure 6A,I; 2.1±0.8 projections/cell, n = 28), decreased in number by the early tailbud stage (Figure 6B,J; 1.5±0.1 projections/cell, n = 12), and were almost entirely absent by the mid tailbud (Figure 6C,K) and late tailbud stages (Figure 6D,L).

This effect in smooth muscle cells is mediated by extracellular TG2 binding to LRP5/6 on the smooth muscle cell surface and this reported interaction does not require transamidation activity.

Tsp4b and Lam bind Itgs (orange) on the muscle cell surface, activating FAK (green) and recruiting Pxn (yellow) and Ilk (purple) to promote muscle specific Itg signaling and stabilize myofiber attachment.

We hypothesized that secreted Tsp4b in the ECM interacts with Itgs on muscle cell surfaces to promote attachment.

A major contributor could be the specific cellular environment in the vasculature since smooth muscle cell surfaces in particular have been shown to promote CAA Aβ aggregation [ 34].

In vascular smooth muscle cells, surface expression of Fas [ 44] and molecules involved in signaling the apoptotic process, such as caspase-8 and caspase-3, appear to be regulated and provide another level of cellular control of apoptosis [ 45].

Here we show that the phenotypes of proliferating tissue progenitor cells such as primary mononuclear muscle cells can also spontaneously fluctuate between different states characterized by the either high or low expression of the muscle-specific cell surface molecule CD56 and by the corresponding high or low capacity to form myotubes.

The homogeneity of the stromal cells that were subsequently transduced were confirmed by real-time reverse transcription-polymerase chain reaction (RT-PCR) using a panel of cell lineage markers (CD24 epithelial, CD45 haematopoietic, von Willebrand factor endothelial, CD146 endothelial, α-smooth-muscle actin [SMA] stromal smooth muscle, and Thy-1 cell surface antigen [CD90] stromal cells).