Sentence examples for muscle cell population from inspiring English sources

One of the advantages of using hiPSCs is that they provide an intact and homogeneous skeletal muscle cell population with almost simultaneous myogenic differentiation in both Control- and DMD-iPStet-MyoD populations.

We assume there are chemotactic mechanisms, like a macrophage colony stimulating factor, that facilitate the migration of smooth muscle cells into the intima to augment the native smooth muscle cell population.

Given the likely contribution of endothelial cells to the total muscle cell population [ 21], it is not surprising that the endothelial cell expression of genes expressed in a number of smaller cell populations may dominate the gene expression signal.

All the transduced cell populations secreted high levels of BMP4 (140–300 ng/106 cells/24 h), but the MDSCs differentiated toward the osteogenic lineage more effectively than did the other muscle cell populations, as indicated by the expression of alkaline phosphatase, an early osteogenic marker.

This could be explained by the following situation: while the satellite cell pool is quickly exhausted by repeated cycles of degeneration and regeneration [ 84], other resident muscle cell populations [ 85] may also contribute to muscle fiber regeneration, along with reinnervation [ 86, 87] in the dystrophic muscle.

In order to analyze the secretome profile of each muscle cells population, we performed cytokine assays.

To monitor the capacity of TNF-alpha to induce insulin resistance (IR) in each muscle cells population, we measured glucose uptake under control conditions and after 24 h TNF-alpha treatment (Fig. 1D).

A differential contribution of cardiac fibroblasts and smooth muscle cells (populations of epicardium-derived cells) to each ventricle may account for part of the morphological-functional disparity.

Similarly, these observations fit the high degree of correlation of the overall expression profiles of all the FSHD and control myogenic samples with each other when compared to the 19 non-muscle cell populations despite the hundreds of significant more-than-two-fold differences in RNA signal for individual genes in comparisons of FSHD and control myoblasts or myotubes.

Cosgrove, B. D. et al. Rejuvenation of the muscle stem cell population restores strength to injured aged muscles.

Based on this work, we hypothesized that the dystrophic phenotype in FRG1 transgenic mice is caused, at least in part, by decreased proliferation in the muscle satellite cell population, which are the cells responsible for maintaining proper muscle regenerative potential.