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Another significant difference in excitability is that heart muscle cannot be tetanized (i.e., induced to spasm) by the application of repetitive stimuli (see above Striated muscle), thus ensuring the completion of the contraction/relaxation cycle and the effective pumping of blood.
However, circadian variations in bladder biophysical properties, including excitability and contractility of bladder smooth muscle, cannot be ruled out entirely.
These results suggest that the relative ATP deficiency in rpS6P−/− muscle cannot be attributed to either a decline in mitochondrial mass or the activity of the measured parameters.
However, the rate of increase in muscle mass, or average daily gain (ADG) of muscle cannot be directly measured on a live animal.
While AMI does not always preclude quadriceps strength gains [ 54], even mild AMI can restrict their magnitude as a portion of the muscle cannot be activated [ 58, 59].
Thus the growth rate of an animal, and more importantly the growth rate of its muscle cannot be accurately estimated from just one measurement.
Similar(48)
There seems to be a tendency towards intrauterine and prenatal cause, but the possibility of a perinatal trauma to the SCM muscles cannot be excluded.
Also adaptations in more proximal muscles cannot be excluded as the origin of the adapted gait.
If fenofibrate increases creatinine outflow from the muscle, muscle damage cannot be ruled out.
Although the positive correlation between muscle mass and muscle strength has been well established [ 2, 7- 11], it is not clear whether higher muscle mass necessarily translates into greater muscle strength or whether gains in muscle strength cannot be achieved without corresponding gains in muscle mass.
The possibility that NLSCELFΔ is less effective at inhibiting CELF activity in skeletal muscle than in heart muscle cells cannot be ruled out.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com