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Laryngeal muscle activation results in diverse changes in vocal fold geometry and mechanical properties; thus, a shift in one parameter alters multiple interdependent parameters simultaneously.
It remains unclear, however, how abnormal lower limb muscle activation results in increased plantar pressures.
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Thirdly, over-active muscle activation resulting in excessive load or stiffness control.
MI of muscle activation resulted in higher isometric force as compared to both MI of muscle relaxation and passive recovery (respectively +2.1%and+3.5%5%).
Secondly, reduced muscle activation resulting in reduced load or stiffness control.
These results indicate that the augmented part of sigh coincided with the genio-glossal (GG) muscle activation, resulting in frequent opening of airway closure with only brainstem or sub-cortical mechanism, but without cortical involvement.
Such reduced muscle activation could result in either poor coordination of movement or reduced arcs of motion.
It is indeed conceivable that variations in muscle activation patterns, resulting from either different locomotor requirements, variations in individual soft tissue competence, or differences in neuromuscular control (24), might influence the manner in which the external moments are balanced by active and passive soft tissue forces between activities and subjects (25).
Similar observations have also been made in skeletal muscle cells: Akt activation results in myofiber growth associated with enhanced Vascular Endothelial Growth Factor (VEGF), a prototypical angiogenic agent, secretion and induces blood vessel recruitment [ 24].
Therefore, more longitudinal studies are needed to analyze the any potential change in the muscle activation pattern as a result of metastability in PT.
In view of the inconsistent results between studies regarding partial diaphragm muscle activation, the degree of diaphragm muscle activity sufficient to preserve diaphragm muscle function or its long-term effects remains to be investigated.
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