Sentence examples for murine kidneys we from inspiring English sources

We were cognizant of this issue; however, as this was one of the first studies that explored sexually dimorphic gene expression changes in human and murine kidneys we opted to maximize the sensitivity at the expense of specificity.

When we compared 67 sex-biased genes in "healthy" human kidneys with 1162 sexually dimorphic genes in the murine kidney (Figure 3A), we identified 9 transcripts that were differentially regulated in both species (corresponding to 13% of all human gender biased genes and 0.8% of all murine gender biased genes).

We and others had reported the presence of endogenous RA in murine kidneys after birth as measured by high performance liquid chromatography (HPLC) [7] [11], which may be synthesized locally by RA synthesizing enzymes (RALDH1-4) tharearexpresseded in the kidney [11] [14].

In summary, here we provide a first description of sex specific gene expression differences in human and murine kidneys under baseline and disease conditions.

It was shown in these and in our studies that the uptake is mainly specific as a consequence of physiologically expressed PSMA in murine kidneys [18, 32].

Injection of MKPC rescued renal damage when administered to IRI damaged murine kidneys, and incorporation of MKPCs into some renal tubules was observed [114].

MRPC potency was demonstrated in vitro, as the cells were able to differentiate into myogenic, adipogenic, neural or osteogenic lineages; further, MRPCs differentiated into renal tubular cells after they were injected into the parenchyma of damaged murine kidneys [113].

Both human and murine kidneys showed bands reacting with the BAMBI antibody.

Only 9 sexually dimorphic transcripts were common to healthy human and murine kidneys and five showed differential regulation in both human and murine diseased kidneys.

Our studies highlight significant differences in human and murine kidneys both at baseline and in their response to injury.

The majority of genes showing gender biased expression either in diseased human and murine kidneys were different from those differentially expressed in healthy kidneys.