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Findings from multivariate analysis, therefore, confirm findings of bivariate analysis for all groups, except for the ideators.
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Thus, age and size are not random and a multivariate analysis is therefore not possible.
Multivariate analysis was therefore performed for patients diagnosed during 1985–1994.
A multivariate analysis was, therefore, carried out to examine their relative contributions in more detail.
In our multivariate analysis we therefore only control for circumcision prevalence.
Multivariate analysis was therefore performed to see whether tumour-associated stroma CC3 expression was independent of these factors and TMN stage and vascular invasion.
For ES also, the effect of main treatment centre differed between 1980 1984 and 1985–1994, and multivariate analysis was therefore restricted to the latter period.
As N status was uncertain in six cases, we omitted these patients from the multivariate analysis, which therefore was performed in 101 patients with all variables available.
A multivariate analysis is therefore required to adjust for these differences, however, account also needs to be taken of the correlation between responses from the same individual.
Third, control-participants were on average younger and had a higher socioeconomic status than did case-participants; however, these differences were addressed in multivariate analysis, and therefore were unlikely to affect validity of results.
In the multivariate analysis model we therefore included the autoantibody titers against oxLDLL, oxLDLP and oxLDLD, and the plasma Lp-PLA2 activity as defined from univariate analysis.
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