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The multiplication of dislocations, severe lattice distortions, and even local amorphization dominate within the dendrites.
In other words, the plastic deformation of the nanotwinned steel is mainly accommodated by glide and multiplication of dislocations.
Cross-slip is shown to be easy and multiplication of dislocations by a double cross-slip mechanism occurs extensively, leading to the formation of large slip bands.
The generation and multiplication of dislocations in an indium phosphide (InP) single crystal grown by the vertical gradient freeze (VGF) process is predicted using a crystallographic model.
In situ and post-mortem diffraction contrast transmission electron microscopy (TEM) was used to study the multiplication of dislocations during a thermal martensitic forward and reverse transformation in a NiTi shape memory alloy single crystal.
We show using in situ SEM and TEM tensile testing of sub-micron Al fibers prepared using selective etching of a eutectic alloy that multiplication of dislocations through intermittent spiral sources directly causes a power-law increase of the yield stress with decreasing cross-sectional size.
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In addition, during deformation, the multiplication of dislocation densities in the MnCu alloy is significantly slower than that in copper, and the transition of the dislocation structure is strongly retarded in the MnCu alloy compared with copper.
These high-resolution observations indicate that, in addition to acting as barriers to slip transmission, CTB can react with a lattice dislocation to facilitate the multiplication of partial dislocations, resulting in translation of the CTB.
One possible model for growth and multiplication of the dislocations in the clusters during solidification where slip on the {1¯10}〈1 1 0〉 system must be allowed is described in detail.
These results can be explained by the mechanism of elastic anisotropy on the dislocation dynamics, as the elastic anisotropy in the MnCu alloy is larger than that in copper, which can strongly retard the multiplication of the dislocation population and the transformation of the dislocation structure.
When the sample is larger than a critical size multiplication of internal dislocation sources provides the dominant mechanism for plastic flow.
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