'multiple virulence factors' is correct and can be used in written English.
It is typically used in scientific and medical contexts to refer to characteristics of a pathogen that allow it to cause an illness or disease. For example, "E. coli can produce multiple virulence factors which are able to cause severe gastrointestinal distress."
Thus, although some isolates from groups A and B1 are limited in their virulence gene content and are not likely to be highly virulent, others in these groups contained multiple virulence factors genes that could contribute to extraintestinal virulence.
Group B Streptococcus (GBS) is a versatile organism which uses multiple virulence factors which bind to the surface of epithelial cells.
S. Typhimurium expresses multiple virulence factors capable of eliciting gut inflammation.
The complex pathology of B. pertussis infection is due to multiple virulence factors having disparate effects on different cell types.
In conclusion, given the multiple virulence factors expressed by S. aureus, a whole cell bacterial vaccine would have significant potential.
Further studies for better understanding of the interaction of different virulence factors at molecular level are necessary as most urovirulent strains express multiple virulence factors simultaneously.
Earlier in vitro studies showed that OMVs derived from Pseudomomas delivered multiple secreted virulence factors into the cytoplasm of airway epithelial cells through a lipid raft-mediated pathway [38].
Our results also show no apparent correlations across the multiple putative virulence factors studied.
It has multiple known virulence factors that contribute to its survival in the oral environment, such as fimbriae, gingipains, lipopolysaccharides (LPS), capsule, and hemagglutinins [ 4].
However, multiple Salmonella virulence factors and homeostatic processes that occur in the macrophage phagosome require disulfide bond formation, indicating that S. enterica has robust mechanisms to regulate redox potential and resist hyperoxidation of thiols in the periplasm (Ellermeier and Slauch, 2004; Miki et al., 2004; Lippa and Goulian, 2012).
The majority of changes in the GAS transcriptome involved down regulation of multiple adhesins and virulence factors and activation of the stress response.
