Sentence examples for multiple tumor cell from inspiring English sources
TRAIL is capable of inducing apoptosis in multiple tumor cell types, with little or no cytotoxicity against normal cells.
Given that Notch controls HIF2α expression in multiple tumor cell types, we investigated whether the level of Notch signaling correlated with HIF2α expression levels across primary cancers, using transcriptome data from multiple tumors of renal, breast, neuroblastoma, and medulloblastoma origin.
The most potent compound 8r showed slightly higher growth inhibitory activity than SAHA in multiple tumor cell lines, even though, antiproliferative activity of 8r seemed inferior to its HDAC inhibition activity.
Among these compounds, the most potent compound 9n exhibited similar if not better HDAC inhibition and antiproliferative activities against multiple tumor cell lines compared with the positive control entinostat (MS-275).
However, direct comparison of multiple tumor cell populations with analysis of the resulting phenotypes of each population within a representative tumor environment has not been clearly described.
The frequent silencing of OPCML-v1 in multiple tumor cell lines and primary tumors but not normal epithelial tissues indicates that OPCML-v1 is likely a tumor suppressor.
Elevated PD-L1 expression on multiple tumor cells is also an important mechanism of tumor-induced immune escape (Iwai et al., 2002; Kataoka et al., 2016).
Bufalin caused the inhibition of growth in multiple tumor cells in a dose-dependent manner, while the results of IC50 revealed that bufalin was more sensitive to U251 and U87 glioma cancer cells than the other tested cancer cells, respectively.
Regarding cytotoxicity study, bufalin caused an obvious inhibition of growth in multiple tumor cells in a dose-dependent manner, while the results of IC50 indicated that bufalin was more sensitive to U251 and U87 glioma cancer cells than the other kinds of cancer cells, respectively.
Stimulation of CD95 on multiple tumor cells induced a conversion from non-CSCs to CSCs.
Abnormal constitutive activation of Tyr705-phosphorylated STAT3 (pSTAT3Tyr705) is observed in multiple tumor cells and contributes to oncogenic processes.
