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After correction for multiple testing, no single single-nucleotide polymorphism was significantly associated with risk of onset TMD.
Four regions were associated with survival (1q21.3 loss p = 0.005, 5p14.1 loss p = 0.004, 9p23 loss p = 0.002, and 15q22.31 gain p = 0.002); however, again, after correcting for multiple testing, no regions were statistically significant, and none were in common with the single marker approach.
After adjusting for multiple testing, no other SNP was associated with clinical sub-types.
Taking into account multiple testing, no linkage/association was found within the regions we identified in this study neither within 10p15 region.
Following Bonferroni correction for multiple testing, no significant associations between the APH1B polymorphism and plasma lipoprotein parameters or other risk factors were observed in the atherosclerosis patients.
After adjusting for multiple testing, no SNP showed significant statistical interaction with any of the non-genetic factors tested (Table S3 and Table S4).
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After correction for multiple tests, no significant correlations were found between BMI and hypocretin-1 autoAbs levels.
After the Bonferroni's correction for multiple tests, no ex post revealed effect remained significant.
After correction for multiple tests, no SNP reached the threshold for statistical significance (Bonferroni significant level P < 1.7 × 10−4).
After correcting for multiple tests, no yeast genes were significant for G. K.ω, (table 2, LRT(4)).
After adjustment for multiple tests, no association between expression of TCF7L2 in eight types of human tissue samples and T2D-associated genetic variants remained significant.
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