Sentence examples for multiple sclerosis suggest from inspiring English sources

Suprasegmental influences on the BR are less well understood, but evidence from studies in patients with movement disorders, stroke and multiple sclerosis suggest input from the central cortex, basal ganglia and thalamus [12].

The findings demonstrate the ubiquity of complement involvement in multiple sclerosis, suggest a pathogenic role for complement contributing to cell, axon and myelin damage and make the case for targeting complement for multiple sclerosis monitoring and therapy.

Recent studies in both rheumatoid arthritis and multiple sclerosis suggest that quantification of type I interferon activity or target gene expression might be informative in predicting responses to distinct classes of therapeutic agents.

The results have implications for the cognitive impairments seen in illnesses characterized by chronic cytokine activation (e.g., Alzheimer's disease) as well as illnesses treated with cytokines (e.g., multiple sclerosis) suggesting that some cognitive failures attributed to working memory impairments per se may better be attributed to prior attention impairments.

This pattern of derepression is observed in some human autoimmune diseases, including multiple sclerosis, suggesting a possible disease association.

However, the success of the anti-α4/β1 integrin antibody in multiple sclerosis suggests that it might be useful in other autoimmune diseases that involve recruitment of T cells.

Moreover, PBLs from patients with relapsing multiple sclerosis had significantly higher miR-326 expression than those from patients with remitting multiple sclerosis, suggesting an association between miRNAs expression and severity of autoimmune response.

However, AQP1 expression is enhanced in reactive astrocytes, accumulating in brain lesions of Creutzfeldt-Jakob disease and multiple sclerosis, suggesting a role of AQP1-expressing astrocytes in brain water homeostasis under pathological conditions.

Recent epidemiological and immunological studies provide evidence for an association between Epstein Barr virus infection and multiple sclerosis, suggesting a role of Epstein Barr virus infection in disease induction and pathogenesis.

Other molecular karyotype and genome sequence studies could not detect any CNV differences between MZ twins that were discordant for cleft lip palate or multiple sclerosis, suggesting that post-zygotic genomic alterations are at least not a common cause of phenotypic discordance.

The ECM staining, similar to that observed in LN T zones, has previously been described in the samples of patients with multiple sclerosis, suggesting that conduits and possibly TRC-like cells may be present in many more sites of chronic inflammation.