Sentence examples for multiple sclerosis pathology from inspiring English sources

Cortical lesions constitute an important part of multiple sclerosis pathology.

Among them, primary and/or secondary alterations in glutamate signalling cause excitotoxicity, which in turn contributes to multiple sclerosis pathology.

These data demonstrate for the first time the relevance of functional anatomical connectivity to the spread of multiple sclerosis pathology in a 'tract-specific' pattern.

A greater understanding of multiple sclerosis pathology is necessary to allow more effective disease management and therapeutic intervention, yielding improved patient outcomes.

Such findings have prompted a reappraisal of the spread of multiple sclerosis pathology, the contribution of lesions and the tissues impacted therein.

In this study, we used the complementary approaches of whole brain post-mortem magnetic resonance imaging and quantitative histology to assess patterns of multiple sclerosis pathology.

In multiple sclerosis, cortical pathology was characterized by the presence of large plaques of primary demyelination (complete loss of myelin with axonal preservation such as preservation of phosphorylated neurofilament-reactive profiles) as described previously (Peterson et al., 2001; Kutzelnigg et al., 2005).

This paper illustrates this issue in one acute neuro inflammatory pathology: Multiple Sclerosis (MS).

A brain biopsy revealed multiple sclerosis (MS -type II pathology.

Inflammation and complement activation are firmly implicated in the pathology of multiple sclerosis; however, the extent and nature of their involvement in specific pathological processes such as axonal damage, myelin loss and disease progression remains uncertain.

During the past 10 years, the intense involvement of the grey matter of the CNS in the pathology of multiple sclerosis has become evident.