Lassmann, H. Hypoxia-like tissue injury as a component of multiple sclerosis lesions.
Han, M. H. et al. Proteomic analysis of active multiple sclerosis lesions reveals therapeutic targets.
Mahad, D., Ziabreva, I., Lassmann, H. & Turnbull, D. Mitochondrial defects in acute multiple sclerosis lesions.
With Dr. Steinman, he worked on microarray profiling of multiple sclerosis lesions.
Mohan, H. et al. Transcript profiling of different types of multiple sclerosis lesions yields FGF1 as a promoter of remyelination.
Waschbisch, A. et al. Analysis of CD4+ Cdouble-positiveTive T cells in blood, cerebrospinal fluid and multiple sclerosis lesions.
Lock, C. et al. Gene-microarray analysis of multiple sclerosis lesions yields new targets validated in autoimmune encephalomyelitis.
The authors report aberrant oligodendrocyte precursor cell (OPC) interactions with blood vessels in certain multiple sclerosis lesions.
Peterson, J. W., Bö, L., Mörk, S., Chang, A. & Trapp, B. D. Transected neurites, apoptotic neurons, and reduced inflammation in cortical multiple sclerosis lesions.
Clemente, D., Ortega, M. C., Arenzana, F. J. & de, C. F. FGF-2 and Anosmin-1 are selectively expressed in different types of multiple sclerosis lesions.
Selmaj, K., Raine, C. S., Cannella, B. & Brosnan, C. F. Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions.
