Sentence examples for multiple pathogens have from inspiring English sources

Multiple pathogens have the potential for interaction resulting in negative impacts on host fitness or alterations in pathogen transmission dynamics.

Together, these results suggest that interactions among multiple pathogens can have important consequences for host health, which may not be predictable from interactions between hosts and individual pathogens.

In the context of infectious diseases, however, less is known about the interplay between autophagy and inflammasome assembly, although it is becoming evident that pathogens have evolved multiple strategies to inhibit and/or subvert these pathways and to take advantage of their intricate crosstalk.

Bacterial pathogens have co-evolved multiple mechanisms to overcome these defenses and to utilize the sequestered micronutrients.

This host-dependent mechanism of transient antibiotic resistance is applicable to multiple pathogens and has implications for the development of more effective antimicrobial therapies.

As genomic approaches are still limited in resolution, there is a possibility that truly shared mechanisms governing susceptibility across multiple pathogen groups have yet to be found.

Licensed live attenuated virus vaccines capable of expressing transgenes from other pathogens have the potential to reduce the number of childhood immunizations by eliciting robust immunity to multiple pathogens simultaneously.

The HGE agent is well established in vector populations in the Northeast (9– 11), and infection with multiple I. scapularis borne pathogens has been documented in both humans and wild mammal reservoirs (9, 11– 11).

The approach of isolating multiple HSPs from stressed pathogens has been extended to bacterial vaccines where broad strain coverage is an advantage, such as in the development of a meningococcal disease vaccine [ 115].

The infection of multiple host species by these pathogens has been implicated as a mechanism that vectors DNA across species boundaries.

Because this resistance is controlled by multiple genes, the pathogen has to undergo multiple mutations to adapt to partial resistance which is more difficult than a single locus mutation.