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Quantitative information on the dynamics of multiple molecular processes in individual live cells under controlled stress is central to the understanding of the cell behavior of interest and the establishment of reliable models.
The nature and outcome of these interactions can be highly diverse, thereby allowing multiple molecular processes to be regulated by lncRNAs.
We recognize that the kinetic processes here must represent the convolution of multiple molecular processes, given the complex oligomeric/polymeric nature of the clusters (see below).
These networks include multiple molecular processes, including the initiation of transcription and epigenetic mechanisms such as methylation of promoters and the post-transcriptional regulation by microRNAs.
We do know that information processing by cells is distributed between multiple intercellular components, such as the cytoskeleton and cell surface receptors, and can depend on multiple molecular processes that occur at different scales.
5, 7 The emerging consensus view is that multiple interacting pathophysiological factors, including protein misfolding, contribute to the neuronal toxicity in ALS. 8, 9 Despite progress in revealing multiple molecular processes involved in disease pathology, relatively little is known about when and how the disekease, which starts focally, spreads throughout the motor network.
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Based on the mass transport resistance network connecting the multiple molecular transport processes in direct flux and surface flux, a theoretical model that captures the pressure dependence of permeation flux is established, offering a possible avenue to predict the mass transport rates through the two-dimensional nanopores.
Examining the dynamics of multiple molecular level processes in a large number of individual cells from the same live cell population under controlled stress allows the construction of statistically meaningful real-time temporal and spatial map of molecular processes necessary to gain deeper understanding of complex signal networks of cellular processes.
This transition from symmetric to asymmetric divisions involves cellular events such as the reorientation of the mitotic spindle and multiple molecular signaling processes including the dynamic regulation of Notch signaling (Egger et al., 2010, 2011).
However, idiopathic pulmonary fibrosis is the result of a complex interaction of multiple cellular and molecular processes.
b) How derangement of multiple, cell-specific, molecular processes can converge on such EEG-visible, correlates of disrupted cognitive function.
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various molecular processes
multiple molecular checkpoints
multiple degenerative processes
multiple molecular regulators
multiple molecular markers
multiple molecular species
multiple developmental processes
multiple molecular mediators
multiple molecular dynamics
multiple molecular pathologies
multiple molecular isoforms
multiple molecular factors
multiple molecular motors
multiple molecular networks
multiple cytoplasmic processes
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